[1] Jang SH.The corticospinal tract from the view point of brain rehabilitation[J]. J Rehabil Med, 2014, 46(3): 193-199. [2] Seo JP, Jang SH.Different characteristics of the corticospinal tract according to the cerebral origin: DTI study[J]. Am J Neuroradiol, 2013, 34(7): 1359-1363. [3] Lemon RN, Griffiths J.Comparing the function of the corticospinal system in different species: organizational differences for motor specialization?[J]. Muscle Nerve, 2005, 32(3): 261-279. [4] Ahn YH, Ahn SH, Kim H, et al.Can stroke patients walk after complete lateral corticospinal tract injury of affected hemisphere?[J]. Neuroreport, 2006, 17(10): 987-990. [5] Jang SH, You SH, Kwon YH, et al.Cortical reorganization associated lower extremity motor recovery as evidenced by functional MRI and diffusion tensor tractography in a stroke patient[J]. Restor Neurol Neurosci, 2005, 23(5-6): 325-329. [6] Cramer SC, Moore CI, Finklestein SP, et al.A pilot study of somatotopic mapping after cortical infarct[J]. Stroke, 2000, 31(3): 668-671. [7] Sawlani V, Gupta RK, Singh MK, et al.MRI demonstration of Wallerian degeneration in various intracranial lesions and its clinical implications[J]. J Neurol Sci, 1997, 146: 103-108. [8] Watanabe H, Tashiro K.Brunnstrom stages and Wallerian degenerations: a study using MRI[J]. Tohoku J Exp Med, 1992, 166(4): 471-473. [9] Lindenberg R, Renga V, Zhu LL, et al.Structural integrity of corticospinal motor fibers predicts motor impairment inchronic stroke[J]. Neurology, 2010, 74(4): 280-287. [10] Koyama T, Tsuji M, Nishimura H, et al.Diffusion tensor imaging for intracerebral hemorrhage outcome prediction: comparison using data from the corona radiata/internal capsule and the cerebral peduncle[J]. J Stroke Cerebrovasc Dis, 2013, 22(1): 72-79. [11] Jayaram G, Stagg CJ, Esser P, et al.Relationships between functional and structural corticospinal tract integrity and walking post stroke[J]. Clin Neurophysiol, 2012, 123(12): 2422-2428. [12] Schaechter JD, Perdue KL, Wang R.Structural damage to the corticospinal tract correlates with bilateral sensorimotor cortex reorganization in stroke patients[J]. Neuroimage, 2008, 39(3): 1370-1382. [13] Yu C, Zhu C, Zhang Y, et al.A longitudinal diffusion tensor imaging study on Wallerian degeneration of corticospinal tract after motor pathway stroke[J]. Neuroimage, 2009, 47(2): 451-458. [14] Liu X, Tian W, Qiu X, et al.Correlation analysis of quantitative diffusion parameters in ipsilateral cerebral peduncle during Wallerian degeneration with motor function outcome after cerebral ischemic stroke[J]. J Neuroimaging, 2012, 22(3): 255-260. [15] Jang SH, Kim K, Kim SH, et al. The relation between motor function of stroke patients and diffusion tensor imaging findings for the corticospinal tract [J]. Neurosci Lett, 2014, 572: l-6. [16] Jang SH, Kim SH, Cho SH, et al.Demonstration of motor recovery process in a patient with intracerebral hemorrhage[J]. NeuroRehabilitation, 2007, 22(2): 141-145. [17] Liu Z, Zhang RL, Li Y, et al.Remodeling of the corticospinal innervation and spontaneous behavioral recovery after ischemic stroke in adult mice[J]. Stroke, 2009, 40(7): 2546-2551. [18] Ueno M, Hayano Y, Nakagawa H, et al.Intraspinal rewiring of the corticospinal tract requires target-derived brain-derived neurotrophic factor and compensates lost function after brain injury[J]. Brain, 2007, 135(Pt4): 1253-1267. [19] Starkey ML, Bleul C, Zorner B, et al.Back seat driving: hindlimb corticospinal neurons assume forelimb control following ischaemic stroke[J]. Brain, 2012, 135(Pt11): 3265-3281. [20] Okabe N, Shiromoto T, Himi N, et al.Neural network remodeling under lying motor map reorganization induced by rehabilitative training after ischemic stroke[J]. Neuroscience, 2016, 339: 338-362. [21] Chisari C, Fanciullacci C, Lamola G, et al.NIBS-driven brain plasticity[J]. Arch Ital Biol, 2014, 152(4): 247-258. [22] Grefkes C, Ward NS.Cortical reorganization after stroke: how much and how functional?[J]. Neuroscientist, 2014, 20(1): 56-70. [23] Stinear CM, Barber PA, Smale PR, et al.Functional potential in chronic stroke patients depends on corticospinal tract integrity[J]. Brain, 2007, 130(Pt1): 170-180. [24] Schulz R, Braass H, Liuzzi G, et al.White matter integrity of premotor-motor connections is associated with motor output in chronic stroke patients[J]. Neuroimag Clin, 2014, 18(7): 82-86. [25] Wang KC, Kim JA, Sivasankaran R, et al.P75 interacts with the Nogo receptor as a co-receptor for Nogo, MAG and OMgp[J]. Nature, 2002, 420(6911): 74-78. [26] Domeniconi M, Cao Z, Spencer T, et al.Myelin-associated glycoprotein interacts with the Nogo66 receptor to inhibit neurite out growth[J]. Neuron, 2002, 35(2): 283-290. [27] Fournier AE, GrandPre T, Strittmatter SM. Identification of a receptor mediating Nogo-66 inhibition of axonal regeneration[J]. Nature, 2001, 409(68): 341-346. [28] Fouad K, Klusman I, Schwab ME.Regenerating corticospinal fibers in the Marmoset (Callitrix jacchus) after spinal cord lesion and treatment with the anti-Nogo-A antibody IN-l[J]. Eur J Neuorsci, 2004, 20(9): 2479-2482. [29] Liebscher T, Schnell L, Schnell D, et al.Nogo-A antibody improves regeneration and locomotion of spinal cord-injuried rats[J]. Ann Neuor, 2005, 58(5): 706-719. [30] Lee JK, Kim JE, Sivula M, et al.Nogo receptor antagonism promotes stroke recovery by enhancing axonal plasticity[J]. J Neurosci, 2004, 24(27): 6209-6217. [31] Lindau NT, Banninger BJ, Gullo M, et al.Rewiring of the corticospinal tract in the adult rat after unilateral stroke and anti-Nogo-A therapy[J]. Brain, 2014, 137(Pt3): 739-756. [32] Wahl AS, Omlor W, Rubio JC, et al.Neuronal repair. Asynchronous therapy restores motor control by rewiring of the rat corticospinal tract after stroke[J]. Science, 2014, 344(6189): 1250-1255. [33] Chen K, Marsh BC, Cowan M, et al.Sequential therapy of anti-Nogo-A antibody treatment and treadmill training leads to cumulative improvements after spinal cord injury in rats[J]. Exp Neurol, 2017, 292: 135-144. [34] Wong EV, David S, Jacob MH, et al.Inactivation of myelin-associated glycoprotein enhances optic nerve regeneration[J]. J Neurosci, 2003, 23(8): 3112-3117. [35] Habib AA, Gulcher JR, Högnason T, et al.The OMgp gene, a second growth suppressor within the NF1 gene[J]. Oncogene, 1998, 16(12): 1525-1531. [36] Ji B, Case LC, Liu K, et al.Assessment of functional recovery and axonal sprouting in oligodendrocyte-myelin glycoprotein (OMgp) null mice after spinal cord injury[J]. Mol Cell Neurosci, 2008, 39(2): 258-267. |